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Geurts, M, Scheijmans, FE, van Seeters, T. Body temperature in acute stroke: relation to stroke severity, infarct size, mortality, and outcome. Non-infectious fever in the neurological intensive care unit: incidence, causes and predictors. Indicators of central fever in the neurologic intensive care unit. Hypothermia and hyperthermia in children after resuscitation from cardiac arrest. Hyperthermia following cardiopulmonary resuscitation. Clinical chameleons: an emergency medicine focused review of carbon monoxide poisoning. Glasgow Coma Scale and laboratory markers are superior to COHb in predicting CO intoxication severity. Hyperthermia in human ischemic and hemorrhagic stroke: similar outcome, different mechanisms. Systemic inflammatory response depends on initial stroke severity but is attenuated by successful thrombolysis. Carbon monoxide poisoning: risk factors for cognitive sequelae and the role of hyperbaric oxygen. Small differences in intraischemic brain temperature critically determine the extent of ischemic neuronal injury. Relation between brain temperature and white matter damage in subacute carbon monoxide poisoning. Brain temperature measured by 1H-magnetic resonance spectroscopy in acute and subacute carbon monoxide poisoning. Outcome of patients with carbon monoxide poisoning at a far-east poison center. Magnitude of temperature elevation is associated with neurologic and survival outcomes in resuscitated cardiac arrest patients with postrewarming pyrexia.
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Grossestreuer, AV, Gaieski, DF, Donnino, MW.
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Acute carbon monoxide poisoning and delayed neurological sequelae: a potential neuroprotection bundle therapy. Cost of accidental carbon monoxide poisoning: a preventable expense. Carbon monoxide poisoning: pathogenesis, management, and future directions of therapy. Initial diffusion-weighted MRI and long-term neurologic outcomes in charcoal-burning carbon monoxide poisoning. Serum neuron-specific enolase and S-100β levels as prognostic follow-up markers for oxygen administered carbon monoxide intoxication cases. Treatment of fever after stroke: conflicting evidence. Hyperthermia is a surrogate marker of inflammation-mediated cause of brain damage in acute ischaemic stroke. Leira, R, Rodríguez-Yáñez, M, Castellanos, M. Google Scholar | Crossref | Medline | ISI Combating hyperthermia in acute stroke: a significant clinical concern. Hyperthermia after cardiac arrest is associated with an unfavorable neurologic outcome. This study carefully emphasizes fastidious control of pyrexia, particularly during the early period after acute CO poisoning. Body temperature measurements, which are easily and noninvasively recorded at the bedside in any facility, help to predict the risk for poor long-term neurological outcomes. Hyperthermia (adjusted odds ratio (aOR) 5.009 (95% confidence interval (CI) 1.556–16.126)) and maximum temperature (aOR 2.581 (95% CI 1.098–6.063)) within the first 24 h after presentation to the emergency department were independently associated with poor long-term neurological outcomes. No significant difference was found in the time of hyperthermia onset within the first day according to the neurological outcomes. Patients with poor long-term neurological outcomes had higher maximum temperatures than patients with good outcomes. The incidence of poor long-term neurological outcomes was significantly higher in the hyperthermia group than in the normothermia group. Hyperthermia (≥ 37.5☌) developed during the first 24 h after presentation in 55 (27.5%) patients, and poor long-term neurological sequelae assessed at 23 months after acute CO poisoning developed in 19.5% of the patients. This retrospective study included 200 patients with acute severe CO poisoning. This study investigated whether hyperthermia within the first 24 h after presentation was associated with long-term neurological outcomes after acute carbon monoxide (CO) poisoning.